Cutting edge: Enhanced IL-2 signaling can convert self-specific T cell response from tolerance to autoimmunity.

نویسندگان

  • Jason Waithman
  • Thomas Gebhardt
  • Gayle M Davey
  • William R Heath
  • Francis R Carbone
چکیده

Naive and memory T cells show differences in their response to antigenic stimulation. We examined whether this difference extended to the peripheral deletion of T cells reactive to self-Ag or, alternatively, the induction of autoimmunity. Our results show that although both populations where susceptible to deletion, memory T cells, but not naive T cells, also gave rise to autoimmunity after in vivo presentation of skin-derived self-Ags. The same migratory dendritic cells presented self-Ag to both naive and memory T cell populations, but only the latter had significant levels of the effector molecule granzyme B. Memory T cells also expressed increased levels of the high affinity IL-2 receptor chain after self-Ag recognition. Provision of IL-2 signaling using a stimulatory complex of anti-IL-2 Ab and IL-2 drove the otherwise tolerant naive T cells toward an autoimmune response. Therefore, enhanced IL-2 signaling can act as a major selector between tolerance and autoimmunity.

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عنوان ژورنال:
  • Journal of immunology

دوره 180 9  شماره 

صفحات  -

تاریخ انتشار 2008